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Ann Thorac Surg 2003;76:688
© 2003 The Society of Thoracic Surgeons

Invited commentary

Ian M. C. Dixon, PhDa, D. H. Freed, MD, PhDa

a Institute of Cardiovascular Sciences, St. Boniface General Hospital Research Center, University of Manitoba, 351 Tache Avenue, Winnipeg MB R2H 2A6 Canada

e-mail: idixon@sbrc.ca

The first 20% of the full text of this article appears below.

The emerging roles of MMPs in the pathogenesis of different forms of cardiovascular disease has garnered increased attention from researchers in both clinical and basic sciences during the past decade. Fibrillar collagen, ie types I and III, confer tensile strength to tissues, and these proteins are constitutively expressed by specific nonmyocyte cells in heart and valves. Healthy cardiac valves depend upon ongoing collagen turnover (mediated by cardiac myofibroblasts) for maintenance of normal tissue structure and function. This phenomenon represents a . . . [Full Text of this Article]






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